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3 edition of Cellular and biochemical aspects in diabetic retinopathy found in the catalog.

Cellular and biochemical aspects in diabetic retinopathy

Colloquium on Cellular and Biochemical Aspects in Diabetic Retinopathy (1978 Paris)

Cellular and biochemical aspects in diabetic retinopathy

proceedingsof the Colloquium on Cellular and Biochemical Aspects in Diabetic Retinopathy, held in Paris (France), 2-3 February, 1978, sponsored by Institut national de la santé et de la recherche médicale

by Colloquium on Cellular and Biochemical Aspects in Diabetic Retinopathy (1978 Paris)

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  • 14 Currently reading

Published by North-Holland Publishing Co. in Amsterdam, Oxford .
Written in English

    Subjects:
  • Diabetic retinopathy -- Congresses.

  • Edition Notes

    Statementeditors F. Regnault, J. Duhault.
    SeriesINSERM symposia / Institut national de la santé et de la recherche médicale -- no.7, INSERM symposia (Institut national de la santé et de la recherche médicale) -- no.7.
    ContributionsRegnault, F., Duhault, J.
    Classifications
    LC ClassificationsRE661.D5
    The Physical Object
    Paginationviii,307p. :
    Number of Pages307
    ID Numbers
    Open LibraryOL21702213M
    ISBN 100720406617, 0720406536

    Although the pathogenesis of diabetic retinopathy has not been clearly elucidated, numerous drugs have been developed based on the current understanding of the complicated and intricate biochemical and pathophysiological aspects of the disease. Current therapy for diabetic retinopathy includes laser photocoagulation, surgery, and metabolic control. The PAα/-β and PAβ/-γ protein levels were also higher in the retina and kidney glomeruli of diabetic mice, respectively. Our results demonstrate, for the first time, that high glucose has direct biological effects on cellular proteasome function, and this modulation might be protective against cellular stress or damage induced by high Cited by:

    Diabetes mellitus is associated with a high prevalence of atherosclerotic and microvascular disease (31). Platelets may play a part in the development of these vascular complications. Hyperag-gregability is frequently reported in diabetes mellitus (1, 2, 7–9, 11, 16, 20, 27).Author: E. Dupuy, P. J. Guillausseau.   Together, these studies leave little doubt that neural retinal defects are among the earliest detectable changes in diabetes. Regardless of whether the initial events begin in blood vessels or neural cells, the clinical stages of diabetic retinopathy manifest cellular, histologic, and functional features of a retinal neuropathy (15,55,56). To the best of our knowledge, there is no evidence that a Cited by:

      Results. The prevalence of cA/G polymorphism in diabetic patients was % (A/A), % (A/G) and % (G/G). In patients with diabetic retinopathy, the prevalence of PDR was significantly higher (p=) in diabetic subjects with the cA/A genotype (%; n=78) compared to those with either the A/G or G/G genotype (%, n=).The prevalence of any other micro and Cited by:   In addition, cellular death has also been reported in diabetic retinopathy, in age-related macular degeneration, and in programmed necrosis of the inflammatory cells, with all of them resulting.


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Cellular and biochemical aspects in diabetic retinopathy by Colloquium on Cellular and Biochemical Aspects in Diabetic Retinopathy (1978 Paris) Download PDF EPUB FB2

Cellular and Biochemical Aspects in Diabetic Retinopathy. Diabetes Nov; 27 (11): Cellular and Biochemical Aspects in Diabetic Retinopathy. Paul F Palmberg. Diabetes Nov27 Professional Books; Diabetes Forecast. ; Diabetes Core Update; ADA's DiabetesPro;Cited by: 9.

Cellular and Biochemical Aspects in Diabetic Retinopathy. Diabetes and The Heart. Show more Book Review. Similar Articles. Navigate. Current Issue; Online Ahead of Print; Scientific Sessions Abstracts; Collections; Archives; Diabetes Print ISSN:Online ISSN.

Get this from a library. Cellular and biochemical aspects in diabetic retinopathy: proceedings of the Colloquium on Cellular and Biochemical Aspects in Diabetic Retinopathy, held in Paris (France), February, [François Regnault; J Duhault; Institut national de. Cellular and Biochemical Aspects in Diabetic Retinopathy Edited by F.

Regnault and J. Duhault Elsevier/North-Holland; Amsterdam, New York viii + pages. Dfl$ By K.W. TaylorAuthor: K.W. Taylor. Cellular and Molecular Mechanism of Diabetic Retinopathy 5 AGEs disturb retinal microvascular homeostasis by overproduction of VEGF through the interaction with receptor of advanced glycation end.

The aim of our study was to know the relationship between serum magnesium with diabetic retinopathy Methods: A total of cases from which 40 cases of each type-I, II diabetes mellitus with and. Author(s): Duhault,J(Jacques); Regnault,François; Colloquium on Cellular and Biochemical Aspects in Diabetic Retinopathy,( Paris, France); Institut national de la santé et de la recherche médicale (France) Title(s): Cellular and biochemical aspects in diabetic retinopathy: proceedings of the Colloquium on Cellular and Biochemical Aspects in Diabetic Retinopathy, held in Paris.

Diabetic retinopathy (DR) is one of the most common microvascular complications of diabetes and remains a major cause of preventable blindness among adults at working age. DR involves an abnormal pathology of major retinal cells, including retinal pigment epithelium, microaneurysms, inter-retinal oedema, haemorrhage, exudates (hard exudates Cited by: Capillary and glial changes during diabetic retinopathy.

Transmission electron micrograph showing a typical normal retinal capillary (A) composed of an endothelial cell (E) and pericyte (P) surrounded by BM (arrows).

In a diabetic rat, the BM is thickened (arrows) (B).Cited by: The AGE hypothesis proposes that hyperglycemia contributes to the pathogenesis of diabetic complications including retinopathy. However, their role in diabetic retinopathy remains largely unknown. This review discusses the chemistry of AGEs formation and their patho-biochemistry particularly in relation to diabetic by: Introduction.

Diabetes mellitus (DM) is a group of chronic diseases marked by high levels of blood glucose resulting from defects in insulin production, insulin use or both. 1,2,3,4,5 The inability of the body to use glucose resulting from the deficiency of insulin leads to constant hyperglycaemia which results in chronic complications, dysfunction and failure of different organs, especially Author: Solani D.

Mathebula. Moreover, the effect sizes of the impacts of dry and wet OAB on QOL were larger than those of diabetic neuropathy or retinopathy, diabetes duration, or urinary tract infection history. Book Review. Diabetes Guidebook, Diet Section.

Diabetes May; 25 (5): Cellular and Biochemical Aspects in Diabetic Retinopathy. Diabetes and The Heart. Show more Book Review. Similar Articles. Navigate. Current Issue; Online Ahead of Print; Diabetes Print ISSN: Diabetic retinopathy, a common complication of diabetes, develops in 75% of patients with type 1 and 50% of patients with type 2 diabetes, progressing to legal blindness in about 5% [60, 61].

As the worldwide prevalence of diabetes continues to increase, diabetic retinopathy is a leading cause of loss of vision in developed countries [ 62 ].Cited by: Diabetic retinopathy (DR) is the most common microvascular complication of diabetes and one of the major causes of used as models to provide valuable information on the cellular and molecular aspects of pathogenesis of DR.

Diabetes in animals is biochemical techniques (such as quantitative PCR, microarray, PDR, Diabetic retinopathy DR Cited by: Retinopathy, a sight-threatening disease, remains one of the most feared complications of diabetes. Although hyperglycemia is the main initiator, progression of diabetic retinopathy continues even after re-institution of normal glycemic control in diabetic patients, and the deleterious effects of prior hyperglycemic insult depend on the duration and the severity of this insult, suggesting a Cited by: Cellular and Molecular Mechanism of Diabetic Retinopathy 15 of glutamate from the extracellular sp ace causing excitotoxicity leading to neurodege neration [Li Q, ; Diederen RM, ].

Diabetic retinopathy is one of the most common devastating complications of diabetes. Currently there a are no accepted drug treatments for diabetic retinopathy and laser therapy is the most accepted treatment option. Biochemical and physiological changes that occur very early in the retina of diabetic patients are the major signaling determinants of future damage to the retina.

Additional biochemical alterations are known to occur in the diabetic retina, but once hypoxia is established, it will cause a cascade of effects that drive the production of VEGF (Figure 16) leading to further changes associated with diabetic retinopathy: and these changes in turn will exacerbate any underlying hypoxia.

Book Review. The Diabetic's Handbook. Diabetes The Diabetic's Handbook. Diabetes Nov9 (6) ; DOI: Related Articles. Cited By More in this TOC Section. Book Review. Cellular and Biochemical Aspects in Diabetic Retinopathy. Diabetes and The Heart. Show more Book Review. Similar Articles.

Navigate. Current Issue. Diabetic retinopathy (DR) is the leading cause of acquired blindness in working age adults worldwide. Biochemical changes in DR contribute to both the microscopic structural and functional changes in the retina.

All these alterations result in macroscopic retinal damage that can be assessed by by: 2.The association of prevalent diabetic retinopathy with serum ascorbic acid and alpha-tocopherol was studied among participants with type 2 diabetes (>or=40 years) (n = ) in the Third National.One of the major complications in patients with diabetes is diabetic retinopathy (DR), a leading cause of blindness worldwide.

It takes several years before any clinical signs of retinopathy appear in diabetic patients, which gives an ample opportunity for scientists to uncover biochemical and molecular mechanism implicated early in the.